【Product】How does resistant dextrin manage blood sugar?

 We know that healthy blood sugar levels are very important to the body's health. Blood sugar fluctuates like a roller coaster and can cause various complications. A stable blood sugar level can maintain the needs of various organs and tissues in the body. Today's introduction starts with how does resistant dextrin manage blood sugar.

Understanding Resistant Dextrin - Product Properties

Resistant dextrin is a low-calorie dextran that is refined and processed from natural starch through hydrolysis, polymerization and other steps. It belongs to low-molecular water-soluble dietary fiber. Announcement No. 16 of the Ministry of Health of China in 2012 has listed resistant dextrin as a common food.

Resistant dextrin is white to light yellow powder, low sweetness (sweetness is about 10% of sucrose), low viscosity (30°C, 30% solution viscosity is about 15cps), low calorie (calorie is about 1.7kcal/g) , no other peculiar smell, soluble in water, heat-resistant, acid-resistant, freeze-resistant, can be used as a low-calorie soluble raw material, applied to low-calorie, low GI, blood sugar control and other foods.

Resistant Dextrin - Blood Sugar Management Effects

Resistant Dextrin helps maintain normal, healthy blood sugar and insulin levels . Compared with ingestion of 50g of glucose, 50g of resistant dextrin induced hypoglycemic response (glucose response = 25) and hypoinsulinemic response (insulin response = 13) ^[1] . Therefore, it can be used as a slow-release energy carbohydrate to partially or completely replace other carbohydrates such as sugar and starch. For example, the syrup made of resistant dextrin is used in concentrated fruit juice drinks, and the glucose response caused by dilution is only 10% of the equivalent sugar products. ^[2]

Ingestion of resistant dextrin- based syrup (18.3 g of resistant dextrin per 100 g based on concentrated fructose syrup ), commercial syrup reference (both products were similarly diluted as specified by the manufacturers), or 50 g of anhydrous glucose ingestion after intake, the average change of human blood glucose concentration. ^[2]

Research Data - How Does Resistant Dextrin Work for Blood Sugar Management

Research data show that resistant dextrin can significantly improve the glucose tolerance of diabetic mice and reduce the rate or amount of glucose absorbed by the body , which may be the mechanism for regulating blood sugar. At the same time, resistant dextrin will not be digested and absorbed in the digestive tract . With the formation of gel , water-soluble resistant dextrin will absorb glucose and slow down the diffusion of sugar , delaying the absorption in the intestine , thereby inhibiting The rise in blood sugar after the absorption of sugar ^[3] .

In addition, prebiotic supplementation has been reported to promote L-cell differentiation in the colon and increase the secretion of gut hormones, including peptide YY, GLP-1, and gastric inhibitory polypeptide ^[4] . Prebiotics may mediate these effects by increasing the bacterial production of butyrate and propionate, thereby activating G protein-coupled receptors, free fatty acid receptor 2, and free fatty acid receptor 3 ^[5] . These hormones help regulate appetite and control glucose metabolism and insulin resistance, respectively. Through these mechanisms, resistant dextrins act as prebiotics to control blood sugar and insulin response.

References

[1] Donazzolo Y, Pelletier X, Cristiani I et al.(2003)Glycemic and insulinemic indexes of NUTRIOSE® FB in healthy subjects. Proceedings of the Dietary Fibre Conference. Noordwijkerhout, The Netherlands, P53.

[2] Lefranc-Millot C, Wils D, Henry J et al. (2006) NUTRIOSE®, a resistant dextrin, and MALTISORB®, a sugar alcohol, two key ingredients for healthy diets and obesity management. Obesity Reviews 7 (Suppl. 2): 269.

[3] Zhang Zesheng, Zhu Jie, Zhang Ying, eta. Effects of resistant dextrin on blood glucose and glucose tolerance in mice [J Food Science and Technology, 2010.35(08): 112-114.

[4]Cani PD, Lecourt E, Dewulf EM, et al. (2009) Gut microbiota fermentation of prebiotics increases satietogenic and incretin gut peptide production with consequences for appetite sensation and glucose response after a meal. Am J Clin Nutr 90, 1236–1243.

[5] Lin HV, Frassetto A, Kowalik EJ Jr, et al. (2012) Butyrate and propionate protect against diet-induced obesity and regulate gut hormones via free fatty acid receptor 3-independent mechanisms. PLOS ONE 7, e35240.